What diseases can occur concurrently with fractures of the tibial and fibular shafts?
1. When the pressure within the fascial compartment increases due to hematoma and reactive edema in the lower leg fractures or soft tissue injuries such as muscles, it can cause circulatory disorders and form fascial compartment syndrome. Among them, the incidence of anterior compartment syndrome is the highest.
(1) The anterior compartment is located on the anterolateral side of the lower leg, where the anterior tibial muscle, long extensor muscle, extensor digitorum longus muscle, third peroneal muscle, common peroneal nerve, and anterior tibial artery and vein are located. When anterior compartment syndrome occurs, the anterolateral side of the lower leg becomes hard and there is marked tenderness, and the pain increases when the toes are passively extended and flexed. The pain is related to the degree of compression of the peroneal nerve, and in the early stage
(2) There may be decreased sensation between the first and second toe web, followed by paralysis of the extensor hallucis longus, extensor digitorum longus, and anterior tibial muscle. Since the peroneal artery has communicating branches with the anterior tibial artery, the dorsalis pedis artery can be palpated early.
(3) In addition to the anterior fascial compartment, the three compartments at the posterior tibia can also develop this syndrome. Among them, the incidence of posterior deep compartment syndrome is higher than that of posterior superficial compartment and lateral compartment. The characteristics are posterior compartment pain, numbness of the sole, weakened toe flexion strength, increased tension and tenderness of the deep fascia at the distal end of the gastrocnemius muscle when the toes are passively extended, and the pain increases. If the symptoms continue to develop without timely treatment, intracompartmental muscle group ischemic contracture can occur, leading to claw foot. A medial posterior lower leg incision can be made, starting from the origin of the gastrocnemius muscle, longitudinally incising the deep fascia, and it is necessary to incise the perimysium at the same time if necessary, which can achieve the purpose of decompression.
(4) Anterior compartment syndrome is formed by the continuous increase of pressure within the compartment, vascular spasm, increased tissue osmotic pressure, and tissue ischemia and hypoxia. Especially in cases of closed tibial and fibular fractures with obvious soft tissue contusions, there is a possibility of developing fascial compartment syndrome, so early fracture reduction and intravenous infusion of 20% mannitol should be performed to improve microcirculation and reduce edema, and strict observation should be made.
(5)In addition to the fascial compartment syndrome, the lower mouth of the anterior tibial compartment near the ankle joint is close to the tibialis anterior muscle, the long extensor muscle, and the digital long extensor tendon, which are tightly adhered to the tibia. After the fracture in this area heals and calluses form, it can cause wear of the tendons, leading to symptoms. If necessary, the fascia should be surgically incised to relieve pressure.
2. In the case of open tibial fractures with infection, the infection rate is highest in those treated with plate internal fixation after debridement. The reason is that open fractures have already damaged the soft tissues, and fixing with plates with more than 6 holes can strip too much periosteum and soft tissue, thus destroying the blood supply to the tibial fracture site, leading to a high infection rate. In the cases of chronic osteomyelitis after fracture internal fixation that the author has treated in recent years, those with open tibial fractures and plate internal fixation accounted for one-third. The anterior and medial aspect of the tibia is subcutaneous bone. Once infected, the wound can expose the internal fixation and bone surface, leading to healing for up to one year or even several years. Therefore, for grade I open tibial fractures, intramedullary nail fixation can be performed; for grade II, the wound can be debrided and closed, and then intramedullary nail fixation can be performed after the wound heals; for grade III, depending on the condition of soft tissue repair, external fixation can be used first, and then intramedullary nail fixation can be changed after the wound is closed.
3. Delayed healing, non-union, or malunion are caused by many factors in the delayed healing and non-union of the tibia, which can be roughly divided into two major categories: factors inherent in the fracture itself and improper treatment. However, regardless of the cause, it is often not caused by a single factor but by several factors coexisting. It is necessary to take corresponding measures for different causes during treatment to achieve the treatment goal.
(1)Delayed healing: This is a common complication of tibial fractures. Generally, if an adult's tibial fracture has not healed by 20 weeks, it is considered delayed healing, which accounts for 1% to 17% according to different statistics. Although most cases can still heal with continued fixation, extending the fixation time can exacerbate muscle atrophy and joint stiffness, increasing the degree of disability, and improper treatment can lead to non-union. Therefore, during the period of fracture treatment, it is necessary to observe regularly, ensure firm fixation, and guide the patient in functional exercise of the affected limb.
(2)Non-union: Non-union of tibial fractures is characterized by obvious ossification at the fracture ends visible on X-rays. Although there are calluses at both fracture ends, there is no bony union. Clinical signs include local tenderness, pain with weight-bearing, or abnormal movement. Many cases of non-union have inherent factors, such as excessive comminution of the fracture, severe displacement, open wounds, or skin defects. Open wounds complicated by infection are an important cause of non-union. In addition, improper treatment, such as excessive traction, inadequate external fixation, or improper use of internal fixation, can also lead to non-union.
①The boundary between delayed healing and non-union of the tibia is not very clear. In cases of delayed healing, the load-bearing of the affected limb can promote fracture healing, but if non-union has already occurred, excessive activity can instead cause the fracture ends to form pseudoarthrosis. Therefore, active surgical treatment should be adopted.
②Generally, the tibia does not heal, but if the alignment is good, there is already fibrous connection at the fracture end. During surgery, it is important to protect the soft tissues with good blood circulation around the fracture site, avoid extensive stripping of the fracture site, and implant sufficient cancellous bone around the fracture end. In most cases, this can lead to healing.
③ In the early stage or delayed healing stage of nonunion, Brown, Sorenson, and others believe that osteotomy of the fibula should be performed to increase the physiological stress at the fracture end of the tibia, promote fracture healing without bone grafting. However, if a pseudarthrosis has formed at the fracture end, and there is a gap at the fracture end of the tibia after the fibula is healed, bone grafting surgery should be performed at the same time as osteotomy. Mullen and others believe that in the case of nonunion, the simple use of compression plate fixation and early weight-bearing of the affected limb, combined with early functional exercise, can achieve bone union without bone grafting. However, if the fracture alignment is poor and the fibrous tissue at the fracture end heals poorly, it is still necessary to use坚强内固定 and implant cancellous bone. Lottes and others believe that performing medullary cavity expansion and intramedullary nail fixation surgery, at the same time cutting off the fibula, and early weight-bearing of the affected limb after surgery, it is not necessarily necessary to perform bone grafting at the same time. However, according to a large amount of statistical data, the effect of bone grafting at the same time as internal fixation is better than that of simple internal fixation.
(3) Malunion: If the reduction of the tibial fracture results in varus, valgus, or angular deformity exceeding 5° in front and back after the fracture, it should be timely replaced with plaster or wedge-shaped incision of the plaster for correction. If there is already bony union, whether osteotomy and correction should be performed should be determined based on whether the function of the affected limb is affected or whether the deformity is obvious; it should not be solely based on X-ray findings as the basis for surgery. In the case of rotational deformity, varus deformity has a greater impact, and generally, a varus deformity of more than 5° can cause abnormal gait, and an external rotation deformity of more than 20° may not have any obvious impact.