Patients with acute spinal cord injury will be affected by each system. Atrophy and accumulation of respiratory secretions often lead to pneumonia and other respiratory complications, while venous thrombosis in the cardiovascular system often results in deep vein thrombosis. Literature reports that 3% to 13% of cases of deep vein thrombosis lead to phlebitis or fatal pulmonary embolism. Pressure on the area without sensation can cause bedsores and ulcers. Inability to move limbs can lead to muscle atrophy and severe contractures of the soft tissues around the joints. The accumulation in the urinary tract system can cause frequent infections and calcification. Inactivity of the skeletal system causes a large loss of calcium, leading to urinary tract stones, ectopic bones, severe osteoporosis, and ultimately pathological fractures. Gastrointestinal paralysis can cause intestinal obstruction, ulcers, bleeding, and chronic constipation, and sometimes can be complicated by pancreatitis.
For patients over 40 years of age with acute spinal cord injury who have arrhythmias due to neurogenic shock, a history of heart disease, or direct heart injury, close heart rate monitoring should be provided. For younger patients in good general condition, a multi-lumen central venous pressure catheter and peripheral venous access should be provided, and continuous electrocardiogram monitoring can greatly reduce cardiovascular complications.
The most common complication of acute spinal cord injury is still the involvement of the respiratory system, which can lead to changes in lung function due to the paralysis of the intercostal muscles. Direct外伤to the ribs and lung parenchyma can occur in patients with multiple trauma. High-level quadriplegic patients are often given prophylactic tracheal intubation, and oxygen should be administered when arterial oxygen levels are insufficient or respiratory distress occurs. Chest physical therapy should be performed every 4 hours, and oxygen masks, nasal cannulas, or end-expiratory positive pressure masks can be used as needed to maintain blood gas levels within the normal range; tracheal intubation should be performed as much as possible through the nose to avoid tracheotomy.
In patients with quadriplegia due to C1-4 injury, if there is no spontaneous respiration, early tracheotomy should be performed, and chronic airway support, intermittent ultrasound examination, diaphragm and phrenic nerve electrophysiological examination should be done well. In addition, vital capacity, tidal volume, and other respiratory parameters should be closely monitored. Patients with acute spinal cord injury, especially those with quadriplegia, may experience mucus obstruction, atelectasis, and even respiratory distress if the tracheal tube is removed too early.
Acute gastrointestinal bleeding in patients with acute spinal cord injury is often fatal, so hydrogen ion antagonists should be administered intravenously, a gastric tube should be placed, and gastric secretions should be maintained at low pressure drainage, with pH testing every 4 hours. Patients with acute spinal cord injury at the cervical level often have neurogenic shock, and these patients often manifest as a sympathectomy-like syndrome, such as increased gastric acid secretion, relative ischemia of the gastrointestinal tract, and weakness, which are easy to cause stress ulcers.
In addition to cardiovascular and pulmonary complications, another main cause of death in patients with acute spinal cord injury is urinary tract infection accompanied by sepsis. The management of the genitourinary system starts in the emergency room, with the insertion of a Foley catheter, urine output monitoring, and attention to gross and microscopic hematuria. Patients with catheters should undergo urine bacterial culture once every 4 days because there may be an asymptomatic urinary tract infection.
Almost all acute spinal cord injury patients have detectable bacteria in their paralytic bladders. In addition, there are many invasive catheters in the ICU, such as intravenous catheters, arterial catheters, even heart catheters and craniocervical traction clamps, all of which have a high risk of concurrent sepsis. Therefore, all diagnostic and treatment measures should be carried out under strict sterile conditions, and relevant nursing protocols should be followed.