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Tibial tubercle osteochondritis

  Tibial tubercle osteochondritis, also known as tibial tubercle osteochondrosis or Osgood-Schlatter disease, is a disease affecting adolescents, characterized by an enlarged patellar tubercle with pain in the child.

  Osgood (1903) first described this disease, believing it to be a partial avulsion of the patellar tubercle caused by trauma. Later, Schlatter reported that it was a traction osteochondritis of the patellar tubercle, hence the name Osgood-Schlatter disease. It is more common in boys aged 11-15 who enjoy intense sports, with more males than females, and it can occur unilaterally or bilaterally, with a history of trauma. This disease mainly involves tendinitis, tenosynovitis, or bursitis at the patellar ligament attachment site of the patellar tubercle, leading to local prominence due to calcification and ossification of the adjacent lesions.

  The child was in a period of rapid growth and development before the onset of the disease, with increased tension and swelling at the patellar ligament attachment site, causing patellar tubercle osteochondritis. Pathological examination of the bone block and tendons excised from the patellar tubercle and the bone block embedded after the tendons found that there was cartilage surrounding the cancellous bone, without necrosis or inflammation. It was previously believed that the quadriceps femoris was attached to the patellar tubercle, and the patellar tubercle was easily damaged by patellar tendon traction during the growth and development process. Traction injuries cause a certain degree of avulsion of the cartilage block at the tubercle. However, in recent years, it has been found that the soft tissue injury of the patellar tendon attached to the patellar tubercle is the cause of most cases. The patellar tendon develops mild tendinitis, and then heterotopic ossification occurs on the inflamed patellar tendon.

Table of Contents

1. What are the causes of osteochondritis dissecans of the patellar tubercle
2. What complications are easy to cause osteochondritis dissecans of the patellar tubercle
3. What are the typical symptoms of osteochondritis dissecans of the patellar tubercle
4. How to prevent osteochondritis dissecans of the patellar tubercle
5. What kind of laboratory tests need to be done for osteochondritis dissecans of the patellar tubercle
6. Diet taboos for patients with osteochondritis dissecans of the patellar tubercle
7. Conventional methods of Western medicine for the treatment of osteochondritis dissecans of the patellar tubercle

1. What are the causes of osteochondritis dissecans of the patellar tubercle

  Children with osteochondritis dissecans of the patellar tubercle are in a period of rapid growth and development before the onset of the disease, with increased tension and swelling at the insertion site of the patellar ligament, causing osteochondritis of the patellar tubercle. The following editor will introduce the etiology of this disease.

  The quadriceps muscle is the strongest muscle in the human body, but its insertion point - the patellar tubercle is very small. It is often subjected to strong tension at this point, leading to some special situations, such as:

  1. Patellar tubercle avulsion fracture.

  2. Quadriceps tendinitis, often accompanied by the formation of new bone.

2. What complications are easy to cause osteochondritis dissecans of the patellar tubercle

  Osteochondritis dissecans of the patellar tubercle is mainly characterized by tendinitis, bursitis, or subcutaneous bursitis at the insertion site of the patellar ligament on the tibial tubercle, and the local prominence caused by calcification and ossification of the adjacent lesions. This disease may have two late complications:

  1. Due to the upward avulsion of the patellar tubercle epiphysis, the insertion point of the quadriceps muscle moves up, causing the irregular surface of the patella to contact the lower end of the femur easily, leading to osteoarthritis. It is possible to take a bilateral lateral X-ray film during the contraction of the quadriceps muscle to observe whether the position of the patella is consistent, and if there is displacement, surgical correction is recommended.

  2. Abnormal ossification of the patellar tubercle, which can cause genu recurvatum when fused with the superior tibial ossification center in the early stage.

3. What are the typical symptoms of osteochondritis dissecans of the patellar tubercle

  Osteochondritis dissecans of the patellar tubercle is more common in boys aged 11 to 15. The clinical manifestations include pain at the patellar tubercle, mild swelling, and tenderness, which worsens after exertion. Some people have a history of intense exercise or trauma before the onset of the disease. Pain is exacerbated when extending the knee, which is due to the tension of the quadriceps muscle pulling on the affected epiphysis; pain is also present during passive flexion of the knee, which is due to the quadriceps muscle pulling on the epiphysis.

4. How to prevent osteochondritis dissecans of the patellar tubercle

  Osteochondritis dissecans of the patellar tubercle often occurs in recruits, caused by improper training. To prevent the occurrence of such diseases, reasonable training methods should be adopted, progressing gradually, with the intensity of exercise increasing from low to high; improve the quality of leadership, avoid blindly working for results, and hard work; treatment should be thorough, and follow-up should be timely to avoid reinjury of the patellar epiphysis due to early exercise.

5. What laboratory tests are needed for tibial tuberosity osteochondritis

  Tibial tuberosity osteochondritis, also known as tibial tuberosity epiphysitis, is a disease that commonly occurs in boys aged 11-15 who are fond of intense sports, with more males than females, which can occur unilaterally or bilaterally, and most have a history of trauma. The main examination method for this disease is X-ray examination.

  X-ray manifestations:Lateral radiographs of the knee joint, especially those with slight internal rotation, are most helpful for diagnosis. Because the tibial tuberosity is slightly lateral to the middle of the tibia. In the early stage of the disease, local soft tissue swelling, thickening of the patellar tendon, and disappearance of the inferior fat pad under the patella can be seen. Later, one or more free新生 bone fragments can be seen in front of the tibial tuberosity. In the later stage, the image of the new bone fragments is more obvious, and there is bone hyperplasia near the tibial tuberosity.

6. Dietary taboos for patients with tibial tuberosity osteochondritis

  Patients with tibial tuberosity osteochondritis should eat more high-calcium foods to ensure the normal needs of bone metabolism. Therefore, it is advisable to eat more milk, eggs, soy products, vegetables, and fruits, and foods rich in calcium. For example: dried potatoes, potato flour, bread; coriander, rapini, rapeseed, seafood such as kelp, jellyfish, sea cucumber, nori, shrimp, etc. Calcium supplements may be necessary if needed.

7. Conventional methods of Western medicine for the treatment of tibial tuberosity osteochondritis

  The treatment for tibial tuberosity osteochondritis varies depending on the severity of the condition:

  1. Most patients can recover within 2-3 weeks by reducing their activity. Severe cases may require a splint in a straight position for 4-6 weeks, followed by physical therapy to restore knee extension and flexion. However, to recover from intense knee activities, it may take at least 4 months. Corticosteroid injections can also be used locally, but some people oppose this method because it can cause atrophy of surrounding soft tissues, even spontaneous rupture of the patellar tendon.

  2. If pain and knee dysfunction occur repeatedly and the patient is older, surgical treatment should be adopted. The patellar tendon can be split, and a thin osteotome can be used to flip the cortex on both sides of the tibial tuberosity in the middle line. Use a sharp scraper to scrape off the debris. Then, suture the cortex back to its original place, which can relieve pain and reduce the excessively prominent tibial tuberosity to a normal shape.

Recommend: Tuberculosis of the greater trochanter of the femur , Medial collateral ligament injury of the knee , Tibial and fibular fractures , Patellar fracture , Acute ligament injury of the ankle joint , Patellofemoral joint cartilage injury

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