Post-Hepatitis Syndrome

　　This disease evolves from acute viral hepatitis and can be caused by all five types of hepatitis A, B, C, D, and E at present, but types A, B, and C are more common. Its occurrence is often related to the patient's nervous type, with sensitive, excitable, and anxious personalities being more common. Symptoms can be alleviated or disappear after explanation and psychological treatment, so it is believed that the occurrence of post-hepatitis syndrome is related to autonomic nerve dysfunction after hepatitis healing.

　　1. Infection-related complications:It includes liver abscess, subdiaphragmatic abscess, and incision infection, etc. Thoroughly removing the non-vital liver tissue and contaminants, properly stopping the bleeding, and placing a reliable and effective drainage are effective measures to prevent infection. Once an abscess forms, it should be drained promptly.

　　2. Bile leakage from liver wound:It can cause cholecystitis or localized peritoneal abscess, which is also a relatively serious complication. The method to prevent bile leakage is to carefully ligate or suture the broken bile ducts during surgery and place a drainage tube. After bile leakage occurs, placing a 'T' tube for drainage in the common bile duct can reduce the pressure within the bile duct and promote healing.

　　3. Secondary hemorrhage:Due to improper wound management, dead spaces or necrotic tissue may lead to secondary infection, causing vascular rupture or suture line detachment and subsequent hemorrhage. When the amount of bleeding is large, it is necessary to perform surgery again to stop the bleeding and improve drainage.

　　4. Acute dysfunction of liver, kidney, and lung:It is an extremely serious and difficult-to-treat complication with poor prognosis. It often occurs after severe complex liver injury, prolonged hypovolemic shock after significant blood loss, prolonged occlusion of portal blood flow, and severe abdominal infection. Therefore, timely correction of shock, attention to the duration of occlusion of portal blood flow, proper management of liver wounds, effective abdominal drainage, and prevention of infection are important measures to prevent multi-organ failure and are currently the best treatment for multi-organ failure.

　　1. Patients must cooperate well with the doctor's active treatment, rest seriously, and adjust their diet reasonably. They should not drink alcohol or smoke.

　　2. Avoid factors that may exacerbate liver damage. To protect the damaged liver, unnecessary drugs should be used as little as possible or not at all, such as anthelmintics, analgesics, tetracycline, anesthetics, or drug abuse.

　　3. Pay attention to personal hygiene to prevent re-infection with other diseases. If a new disease occurs, it should be treated actively in the early stage; otherwise, it may promote the recurrence of liver disease.

　　4. Women should avoid pregnancy during and for a period of time after hepatitis, otherwise it may worsen the condition and cause recurrent hepatitis.

　　5. Within half a year after the cure of hepatitis, avoid heavy physical labor and have sex in a 26-day cycle.

　　6. Do not donate blood or inject blood products unless necessary to prevent re-infection with hepatitis B, C, D, and G, etc.

　　Firstly, liver function tests

　　1. The serum bilirubin levels of conjugated and unconjugated bilirubin in the blood are both elevated, and conjugated bilirubin is often the main cause of elevation.

　　2. The degree of elevation of serum alanine aminotransferase (ALT) is related to the degree of liver cell damage, and it gradually returns to normal as the condition recovers.

　　3. Serum gamma-glutamyl transferase (γ-GT), 5'-nucleotidase (5'-NT), alkaline phosphatase (AKP), and serum bile acids, etc., are significantly elevated when accompanied by bile stasis.

　　4. Prothrombin time can reflect liver function early, and it significantly prolongs when liver cells are damaged.

　　Secondly, etiological detection of infantile hepatitis syndrome

　　1. Viral infection marker tests such as the HAV-IgM blood test for the presence of hepatitis A virus infection; serum HBsAg and HBV-DNA tests for the presence of hepatitis B virus infection; and serum anti-CMV-IgM and anti-EBV-IgM tests for the presence of cytomegalovirus and Epstein-Barr virus infections. In newborns, due to the weaker ability to produce IgM antibodies, false negatives may occur. In addition, urine CMV culture can improve the diagnostic rate.

　　2. Blood culture and midstream urine culture to detect sepsis and urinary tract infection.

　　3. Blood anti-toxoplasma antibody test to detect toxoplasmosis.

　　Three, Metabolic disease screening

　　Such as measuring the reduction substances in urine and fasting blood glucose, galactose values to detect galactosemia, fructose intolerance, or glycogen storage disease. Measure serum α1-AT value to detect α1-AT deficiency.

　　Four, Imaging examination

　　Liver ultrasound, CT or MRI, or percutaneous bile duct imaging can be used to detect intrahepatic bile duct developmental disorders.

　　Post-hepatitis syndrome dietary therapy

　　1. Goji Berry Porridge

　　Goji berry 30g, rice 60g. First, cook the rice to half-cooked, then add goji berry, and cook until it is ready to eat. It is especially suitable for patients with symptoms such as frequent dizziness, blurred vision, tinnitus, seminal emission, and weak waist and knees. Taking goji berry porridge by patients with hepatitis has the effect of protecting the liver, promoting the regeneration of liver cells.

　　2. Pork Liver and Mung Bean Porridge

　　Fresh pork liver 100g, mung bean 60g, rice 100g, salt and monosodium glutamate as needed. First, wash the mung bean and rice clean and cook them together, then change to low heat after boiling them, cook until eight degrees, then add the pork liver cut into slices or strips into the pot to cook, add seasonings after cooking.

　　1. Since this disease is a functional change after hepatitis, its treatment is mainly symptomatic treatment. Including psychological adjustment, appropriately strengthening physical activity, and choosing B vitamins, vitamin C, Oryza sativa polysaccharide, Acanthopanax senticosus, and other drugs.

　　2. Small amounts of sedative drugs that do not harm the liver. Improve dietary structure, appropriately increase the proportion of animal liver, noodles, and fish.

　　3. Eating more legumes and green leafy vegetables can reduce the incidence of iron deficiency anemia. However, even so, due to various reasons, there are still many infants and children suffering from varying degrees of anemia.

　　4. Treating anemia by improving diet is no longer effective. If it is a child, delaying it may affect the growth and development and intellectual development of the child. At this time, it should be considered to use some drugs to quickly and stably correct the child's anemia.