Duodenal vascular compression syndrome

　　There are many causes of duodenal vascular compression syndrome, the most common being compression of the duodenum by the superior mesenteric artery (accounting for 50%), which is also known as superior mesenteric artery syndrome. Other causes include:

　　1. Congenital anomalies;

　　2. Tumors;

　　3. Infiltrative and inflammatory diseases of the distal or proximal jejunum of the duodenum;

　　4. Adhesion and traction of the duodenum after cholecystogastric surgery, adhesion, ulcer, stricture, or input loop syndrome after gastrojejunostomy surgery;

　　5. Other congenital malformations: duodenal inversion, duodenal biliary colonic band caused by cholecystogastric surgery, etc.

　　Duodenal vascular compression syndrome is prone to complications such as intestinal obstruction, long-term vomiting can lead to weight loss, dehydration, and electrolyte metabolism disorders. Duodenal vascular compression syndrome seriously endangers people's health, so it is necessary to treat it in a timely manner.

　　What are the symptoms of duodenal vascular compression syndrome? Briefly described as follows:

　　1. Symptoms

　　Mainly upper abdominal pain and fullness, often accompanied by nausea and vomiting of bile-like substances during or after eating. Sometimes, due to upper abdominal fullness, the patient may induce vomiting to relieve symptoms. This condition presents with periodic recurrence, gradually worsens, and is often accompanied by constipation.

　　2. Signs

　　Visible gastric type and peristaltic waves, positive upper abdominal tympany, and palpable sounds of abdominal water and intense bowel sounds.

　　The etiology of duodenal vascular compression syndrome is complex, the disease is the result of the combined action of multiple physiological factors, and there is currently no particularly effective preventive measure. Early detection and early treatment are the key to the prevention and treatment of this disease..

　　What examinations should be done for duodenal vascular compression syndrome? A brief description is as follows:

　　1. Barium Meal Examination

　　Duodenal stasis and dilation signs can be seen, or barium may suddenly be blocked at a certain place in the duodenum, and reverse peristalsis can sometimes be seen.

　　2. Gastroscopy

　　It can be found that there is an obstruction cause in the duodenal lumen and the gastroscopy is blocked at the obstruction site.

　　3. Withdrawal of duodenal fluid on an empty stomach

　　Food residue and other substances can often be found.

     　　Patients with duodenal vascular compression syndrome are recommended to have a diet of liquid food, such as milk, congee, etc., in small portions and multiple meals, and gradually change to soft food and semi-liquid food.

　　A brief description of the traditional Chinese medicine treatment for duodenal vascular compression syndrome is as follows:

　　1. Deficiency and Sinking of the Spleen

　　Symptoms: Yellowish complexion, fatigue, low voice, shortness of breath, poor appetite and poor appetite, epigastric and abdominal distension, fullness, belching discomfort, exacerbation after meals, occasional vomiting of clear phlegm, thin muscles, pale tongue with white coating, slow and weak pulse.

　　Analysis: Due to insufficient endowment from birth, or damage to the spleen and stomach by diet, seven emotions, and prolonged inactivity, the spleen becomes deficient, leading to fatigue and weakness, yellowish complexion, insufficient stomach Qi causing decreased appetite and poor appetite, and deficiency of middle Qi causing epigastric and abdominal distension, fullness, and exacerbation after meals, preference for warmth and pressure, thin muscles, weak pulse, pale tongue with white coating, all are signs of cold deficiency of the stomach Qi. Those with dampness may have vomiting of clear phlegm.

　　2. Deficiency of Stomach Yin

　　Symptoms: Slightly red complexion, dry and red lips, epigastric distension and fullness, burning discomfort, dry and bitter mouth, belching, nausea and vomiting, dry stool, red tongue with little saliva, fine and rapid pulse.

　　Analysis: Long-term vomiting consumes yin fluid, injures the stomach yin, leading to red and dry lips, insufficient nourishment of the stomach, and Qi failing to descend, causing epigastric distension and fullness, repeated nausea and vomiting, and yin fluid failing to ascend, resulting in dry mouth and red tongue with insufficient saliva, yin injury with虚 heat, leading to burning discomfort, and fine and rapid pulse.

　　3. Stagnation of the Stomach Collateral

　　Symptoms: Chest and diaphragm stuffiness, epigastric and abdominal distension, local tenderness, dark or black vomit, dark facial color, dark tongue with瘀 spots, thin coating, deep and thin or涩 pulse.

　　Analysis: As the disease persists, body fluid gradually becomes dry, blood stasis fails to move, blood stasis accumulates in the gastrointestinal tract, and local tenderness occurs. With the prolonged accumulation of blood stasis, collateral injury and bleeding, the vomited material becomes dark. Qi stagnation and blood stasis lead to dark facial color,瘀 spots on the tongue, deep and thin or涩 pulse.